Download Acute Renal Insufficiency Made Ridiculously Simple by Carlos Rotellar PDF

By Carlos Rotellar

A quick, transparent, sensible, and funny method of acute renal insufficiency.

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Extra info for Acute Renal Insufficiency Made Ridiculously Simple (MedMaster Series, 2005 Edition)

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X- RA~> Figure 37 Mannitol infusion as prophylaxis. TREATMENT PERIOD I (The Kidney is in Danger) This period starts at the time the kidney receives the insult and continues until intrinsic acute renal failure develops. The length of this period varies from one patient to another. During this time therapeutic 37 intervention may reverse and/or diminish the severity of the ARF (Fig . 38). s. ). It is unclear by which mechanism these measures may change the course of the ATN. In some circumstances the patients may respond to the use of diuretics with an increased urine output, but without a significant increase in the clearance of toxins.

Diffusion transfer is a passive transfer of solutes across a membrane, in the absence of net solven t transfer. Peritoneal Dialysis. Performed continuously with hourly exchanges as needed. CAVH. (Continuous arteriovenous hemofiltration). Continuous blood « 100 ml/rnin) filtration through a high permeability membrane to accomplish ultrafiltration rates between 200-800 ml/hr, Requires continuous fluid replacement and does not need dialysis fluid. This technique is based on the physical principle of filtration and it can be performed continuously (twenty four hours a day, seven days a week).

Fig. 41). Furthermore, we should avoid an excessive intake of free water (water without salt). Potassium Balance Plasma potassium increases quite rapidly in patients with ATN. Hyperkalemia can cause cardiac arrest, therefore , it must be closely followed . Potassium intake should be lower than 40-60 mEq/24 hr; cation exchange resins (Kayexalate) (1) (Fig . 42) and dialysis should be used as needed. v, bicarbonate shift K+ from the extracellular to the intracellular space. Calcium, Phosphate and Magnesium Balance Hypocalcemia, hyperphosphatemia and hypermagnesemia develop in acute renal failure.

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